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Transduction of Voltage and Ca2+ Signals by Slo1 BK Channels
Department of Physiology, The University of Pennsylvania, Philadelphia, Pennsylvania, USA.
Division of Molecular Medicine, Department of Anesthesiology, University of California, Los Angeles, USA.
Division of Molecular Medicine, Department of Anesthesiology, and Brain Research Institute, and Cardiovascular Research Laboratories, University of California, Los Angeles, USA.
2013 (English)In: Physiology (Bethesda), ISSN 1548-9213, Vol. 28, no 3, p. 172-189Article, review/survey (Refereed) Published
Abstract [en]

Large-conductance Ca2+- and voltage-gated K+ channels are activated by an increase in intracellular Ca2+ concentration and/or depolarization. The channel activation mechanism is well described by an allosteric model encompassing the gate, voltage sensors, and Ca2+ sensors, and the model is an excellent framework to understand the influences of auxiliary β and γ subunits and regulatory factors such as Mg2+. Recent advances permit elucidation of structural correlates of the biophysical mechanism.

Place, publisher, year, edition, pages
Amercian Physiolocial Society , 2013. Vol. 28, no 3, p. 172-189
National Category
Biophysics
Identifiers
URN: urn:nbn:se:liu:diva-162181DOI: 10.1152/physiol.00055.2012OAI: oai:DiVA.org:liu-162181DiVA, id: diva2:1371972
Available from: 2019-11-21 Created: 2019-11-21 Last updated: 2019-11-26Bibliographically approved

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Pantazis, Antonios

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